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Lorne Mendell
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Lorne Mendell, Ph.D. is a [[Neuroscientist|neurobiologist]] currently employed as a distinguished professor in the department of neurobiology and behavior at [[Stony Brook University]] in [[New York (state)|New York]].<ref name=":0" /> His research focuses primarily on neurotrophins in neonatal and adult mammals, and on the neuroplasticity of the mammalian spinal cord. [[Neurotrophin|Neurotrophins]] are proteins in a family known as [[Growth factor|growth factors]]. These proteins promote the development, functioning, and neuron survival. [[Neuroplasticity]] refers to a neuron's ability to form new [[neural]] connections; this allows the [[brain]] to learn and memorize and adjust its processes in order to adapt to adverse changes like injury or [[disease]]. Dr. Mendell's research interests lie in other areas including [[pain]], nerve wind-up, and specifically the neurotrophin, [[NT-3]]. Dr. Mendell is a veteran in the neuroscientist; he has held numerous positions throughout his career.<ref name=":0" /> His research has contributed findings to the growing pool of knowledge of [[Axon|axonal]] development and regeneration of immature and mature neurons. Recently, Dr. Mendell has been a part of the search for novel treatments for [[spinal cord]] injuries, and presently Mendell continues to study neurotrophins to determine their effects on neuronal plasticity. Mendell served a term as the President for the Society of Neuroscience during the years 1997-1998.
== Biography ==
=== Life and Education ===
Lorne Mendell graduated from [[McGill University]] in 1961 with a bachelor of science in both [[Mathematics]] and [[Physics]]. He went on to earn his Ph.D. from [[Massachusetts Institute of Technology]] (M.I.T.) in 1965. Shortly after earning his Ph.D., Mendell became a faculty member at [[Duke University Medical Center]] in 1968. He remained at Duke until 1980 when he joined the faculty at Stony Brook University. Since then, Dr. Mendell has held numerous positions in a number of associations and organizations. He has also received many honors and awards throughout his career as a neurobiologist. See Positions Held and Honors & Awards for more information.<ref name=":0"> Renaissance School of Medicine at Stony Brook University|website=renaissance.stonybrookmedicine.edu|access-date=2020-03-29}}</ref>
=== Family ===
Dr. Lorne Mendell is married to Dr. Nancy Mendell, a professor emerita in the department of applied mathematics and statistics at Stony Brook University. Together they have a daughter, Stephanie Mendell. In November 2016, Stephanie Mendell married David Hodgson, son of Mr. and Mrs. Stephen Hodgson.<ref></ref>
== Career ==
=== Research focus/expertise ===
* Neurotrophins
As mentioned before, [[Neurotrophin|neurotrophins]] help promote the [[development]], functioning, and survival of [[Neuron|neurons]]. Mendell's research within neurotrophins has focused specifically on [[Nerve growth factor|nerve growth factors]] (NGF) and their role that they play within inflammatory pain<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref>. Within this, there was also a focus in [[Brain-derived neurotrophic factor|Brain Derived Neurotrophic Factor]] (BDNF) and their sensitizing effect on specific [[synaptic transmission]] between [[Nociceptor|nociceptors]] and their target.<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref> In his recent labs, he has been studying the contributions of a third [[Neurotrophin-3|neurotrophin]] (NT-3) and what the effects are when the [[synapse]] that is involved in [[transmission]] between [[Stretch receptor|stretch receptors]] and [[Motor neuron|motor neurons]] is altered.
* Neuronal Plasticity
[[Neuronal plasticity]] is a very important aspect in the normal functioning of the [[brain]]. It is what allows the brain to continually adjust in light of new situations. This means that neurons within the brain are changing, especially during [[childhood]] when the brain is still developing. Plasticity does occur into [[Adult|adulthood]], just on a reduced scale. This property of the brain has been found to be especially helpful after a brain [[injury]], during [[Physiology|physiological]] learning, etc. The magnitude of neuronal plasticity can range from microscopic, affecting only individual neurons, all the way up to macroscopic, where things such as [[cortical remapping]] may be observed. The purpose of this is to generate a more effectively functioning brain.
Mendell has focused a lot of his research on neuronal plasticity, specifically within the [[Spinal cord|mammalian spinal cord]]. He has combined some of his research from NT-3 to help show how neurotrophins can help alter the function of both damaged and undamaged neurons.
* Pain
[[Pain]] is a physical feeling that most of the time harms an individual. This is caused by a damaging [[Stimulant|stimuli]] that can vary in intensity. The stimuli sends signals that then travel along neurons so that [[information]] can be received by the brain. The nerves that are typically responsible for pain sensation are [[Nociceptor|nociceptors]].
A lot of the work Mendell has done with both [[Neurotrophin|neurotrophins]] and [[Neuroplasticity|neuronal plasticity]] has some overlap into the realm of pain, and what their roles are in both reducing and relieving pain. For example, Mendell has conducted [[research]] which has shown the relationship between [[Nerve growth factor|nerve growth factors]] (a type of neurotrophin) and inflammatory pain.
* Nerve Wind Up
Mendell discovered the property of 'wind up' in the [[spinal cord]]. 'Wind Up' is the property of [[Group C nerve fiber|C-fibres]] in the [[Peripheral nervous system|peripheral nerves]] of the spinal cord that when stimulated at repetitious low frequencies there will be a gradual accumulation in the amplitude of response.<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref>
* NT-3
Mendell has been interested in the role [[Neurotrophin|neurotrophins]] play in nervous [[Neuroplasticity|plasticity]] since at least the mid 1990s. At the time, neurotrophins were understood to be significant factors in neuronal development and [[Differentiation (biology)|differentiation]] in the [[Central nervous system|central]] and [[peripheral nervous system]], however; scientists were still exploring the specific functions of individual neurotrophins. Subsequent findings suggested that neurotrophins have effects on both damaged and undamaged neurons. As such, neurotrophins are a major facet of [[spinal cord injury]] research. Dr. Mendell's research on neurotrophins has evolved synchronously with the understanding of neurotrophins. In more recent times, Dr. Mendell's research has been focused partly on spinal cord injury and the
* Nerve Growth Factor and Hyperalgesia
[[Hyperalgesia]], increase of sensitivity to a stimulus, was found by Mendell to be influenced by the introduction of [[nerve growth factor]], NGF. It functions by interactions with sensory [[Nociceptor|nociceptive]] neurons. Mendell was able to investigate how [[Rodent|rodents]] and [[Mammal|mammals]] sensitivity to thermal and mechanical stimulus was increased through exposure to NGF.<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref> NGF was shown by Mendell to also play a role in the development of young mammals. Mature animals, he showed, that NGF had a possible role in the linkage of [[hyperalgesia]] and [[inflammation]].<ref></ref>
== Major works/Contributions ==
In 1965, Mendell's doctoral dissertation was published in the academic journal [[Nature (journal)|Nature]]. The dissertation was about how [[Group C nerve fiber|C fibres]] in the dorsal horn affect the A fibres. He concluded that his evidence did not support the idea that the firing of C fibres had an effect on the presynaptic hyperpolarization of the A fibres of the cats that were experimented on.
In April 2016, Mendell helped to publish a paper<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref> that focused on the relationship between a protein called [[CD2AP]] and the plasticity of [[Neuron|neurons]]. It is known that the growth of an [[axon]] of a neuron helps with both the adaptive and maladaptive [[Neuroplasticity|plasticity]] within the [[nervous system]]. Therefore, if an axon becomes injured, or cannot function to its best ability, the consequences can be in the form of [[Neurological disorder|neurologic disease]]. The protein CD2AP in this study was looked at in depth, which is responsible for coordinating axon outgrowth. Mendell and his team were able to observe that CD2AP is amplified within [[Dorsal root ganglion|dorsal root ganglions]] (DRGs) during axonal sprouting, but decreases in quantity during axonal [[Regeneration (biology)|regeneration]]. Based on these findings, it is still undetermined what the exact relationship between the control of the regeneration of previously damaged axons and the additional development of non-damaged axons is.
In another one of his studies<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref>, conducted in July of 2011, Mendell et al. looked at the mechanisms involved in the relationship between [[Nerve growth factor|neuronal growth factors]] (NGF), which is a [[neurotrophin]], and pain. It is known that specific mutations in NGF or [[Tropomyosin receptor kinase A|TrkA]] genes can lead to a decrease in the sensitivity to [[pain]]. However, the NGF-TrkA axis, on the other hand, can facilitate the development of many different types of long-term acute and [[chronic pain]]. With this, this study was able to conclude that one has to look at how many NGF-responsive (meaning how many are TrkA-positive) [[Nociceptor|nociceptors]] are actually connected to the tissue where the pain is coming from. This is an important relationship because the amount of [[Nerve|innervation]] widely varies between different types of tissue. Then, once this has been examined, one can more effectively choose which chronic pain state to target. Therefore, if it is able to be applied effectively, choosing therapies that rely on targeting NGF-TrkA signaling may represent a new way to treat different types of chronic pain.
Mendell has also put forth research<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref> about the increased possibility of promoting [[spinal cord]] repair through a combination of several different [[Treatment|treatments]]. The mechanisms involved in this are due to the additive effects when several different neurotrophins and [[Protein|proteins]] are combined, including [[Chondroitin ABC lyase|chondroitinase ABC]], [[Neurotrophin-3|NT3]], and enhanced levels of [[NR2D]]. These together allow for either the synthesis or strengthening of spinal circuits, as well as slight recovery of function for those that have been damaged.
=== Selected Literature for continued reading on Mendell's Research. ===
**https://ift.tt/3bz3T88 (Abstract)
**https://ift.tt/33WsAch (Full Article)
**https://ift.tt/3dIVaCv (Abstract)
**https://ift.tt/2WWFRAc (Full Article)
**https://ift.tt/39u0ypB (Full Article)
**https://ift.tt/2QZRXV8 (Abstract)
**https://ift.tt/2UQV0jw (Abstract)
**https://ift.tt/2UHxFAM (Abstract)
**https://ift.tt/2UvQh8k (Full Article)
**https://ift.tt/2xz9UDe (Abstract)
**https://ift.tt/2WWO2ME (Abstract)
**https://ift.tt/2UHxGVm (Abstract)
**https://ift.tt/33W3yKe (Abstract)
**https://ift.tt/2Uyp58Z (Abstract)
**https://ift.tt/2JsLXQM (Abstract)
**https://ift.tt/2Jx1P4x (Abstract)
**https://ift.tt/2wP9M2u (Abstract)
**https://ift.tt/2WSvVrj (Full article)
**https://onlinelibrary.wiley.com/doi/abs/10.1111/j.1460-9568.2010.07392.x (Abstract)
**https://ift.tt/3dGNKPU (Full Article)
**https://ift.tt/3bH2OeS (Full Article)
**https://ift.tt/3bDamPt (Abstract)
**https://ift.tt/2vZo6F5 (Full Article)
**https://ift.tt/33ZHv5f (Full Article)
**https://ift.tt/2Uu2IRH (Full Article)
**https://ift.tt/2WSvWvn (Full Article)
**https://ift.tt/3augUQk (Abstract)
**https://ift.tt/39BuzUI (Full Article)
**https://ift.tt/39upCNa (Abstract)
**https://ift.tt/2QZ2joq (Abstract)
**https://ift.tt/2UNAd0p (Abstract)
**https://ift.tt/2ymTAWD (Full Article)
**https://ift.tt/2WTHv5u (Full Text)
**https://ift.tt/33XVXL7 (Full Article)
**https://ift.tt/3dISGnC (Abstract)
**https://ift.tt/39vWwx0 (Abstract)
=== Positions Held<ref> Renaissance School of Medicine at Stony Brook University|website=renaissance.stonybrookmedicine.edu|access-date=2020-03-29}}</ref> ===
Chair of the Department of Neurobiology and Behavior, [[Stony Brook University]], 1986-2006
[[Journal of Neurophysiology]]'s Editor-in-Chief, 1983-1989
On the Editorial Board for the [[Journal of Neurophysiology]], current
On the Editorial Board for the [[The Journal of Neuroscience|Journal of Neuroscience]], 1983-1989
On the Council for the [[Society for Neuroscience]], 1986-1990
President of the Association of [[Neuroscience]] Departments and Programs, 1991-1992
Treasurer of the [[Society for Neuroscience]], 1996-1997
President of the [[Society for Neuroscience]], 1997-1998
[[Board of directors|Board of Directors]] for the Craig Neilsen Foundation, 2009
Chairman for the [[Spinal Cord Injury|Spinal Cord Injury Research Board]] of the State of New York, 2013
Senior Editor for [[Neuroplasticity]] for the [[University of Oxford|Oxford University]] Press Research Encyclopedia in Neuroscience, 2015
=== Honors & Awards<ref> Renaissance School of Medicine at Stony Brook University|website=renaissance.stonybrookmedicine.edu|access-date=2020-03-29}}</ref> ===
Distinguished Professor of Neurobiology and Behavior at [[Stony Brook University]]
Selected to serve on the London (UK) Pain Consortium Advisory Board, 2009-2014
Chosen to give the [[History of neuroscience|History of Neuroscience]] plenary lecture ("The Emergence of Contemporary Pain Neuroscience") during the Annual Meeting of the Society for Neuroscience, 2012
Selected to be a part of the International Science Advisory Council of Brain Canada, 2014
Named a Fellow for the [[American Physiological Society]] Inaugural Class, 2015
<br />
== Biography ==
=== Life and Education ===
Lorne Mendell graduated from [[McGill University]] in 1961 with a bachelor of science in both [[Mathematics]] and [[Physics]]. He went on to earn his Ph.D. from [[Massachusetts Institute of Technology]] (M.I.T.) in 1965. Shortly after earning his Ph.D., Mendell became a faculty member at [[Duke University Medical Center]] in 1968. He remained at Duke until 1980 when he joined the faculty at Stony Brook University. Since then, Dr. Mendell has held numerous positions in a number of associations and organizations. He has also received many honors and awards throughout his career as a neurobiologist. See Positions Held and Honors & Awards for more information.<ref name=":0"> Renaissance School of Medicine at Stony Brook University|website=renaissance.stonybrookmedicine.edu|access-date=2020-03-29}}</ref>
=== Family ===
Dr. Lorne Mendell is married to Dr. Nancy Mendell, a professor emerita in the department of applied mathematics and statistics at Stony Brook University. Together they have a daughter, Stephanie Mendell. In November 2016, Stephanie Mendell married David Hodgson, son of Mr. and Mrs. Stephen Hodgson.<ref></ref>
== Career ==
=== Research focus/expertise ===
* Neurotrophins
As mentioned before, [[Neurotrophin|neurotrophins]] help promote the [[development]], functioning, and survival of [[Neuron|neurons]]. Mendell's research within neurotrophins has focused specifically on [[Nerve growth factor|nerve growth factors]] (NGF) and their role that they play within inflammatory pain<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref>. Within this, there was also a focus in [[Brain-derived neurotrophic factor|Brain Derived Neurotrophic Factor]] (BDNF) and their sensitizing effect on specific [[synaptic transmission]] between [[Nociceptor|nociceptors]] and their target.<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref> In his recent labs, he has been studying the contributions of a third [[Neurotrophin-3|neurotrophin]] (NT-3) and what the effects are when the [[synapse]] that is involved in [[transmission]] between [[Stretch receptor|stretch receptors]] and [[Motor neuron|motor neurons]] is altered.
* Neuronal Plasticity
[[Neuronal plasticity]] is a very important aspect in the normal functioning of the [[brain]]. It is what allows the brain to continually adjust in light of new situations. This means that neurons within the brain are changing, especially during [[childhood]] when the brain is still developing. Plasticity does occur into [[Adult|adulthood]], just on a reduced scale. This property of the brain has been found to be especially helpful after a brain [[injury]], during [[Physiology|physiological]] learning, etc. The magnitude of neuronal plasticity can range from microscopic, affecting only individual neurons, all the way up to macroscopic, where things such as [[cortical remapping]] may be observed. The purpose of this is to generate a more effectively functioning brain.
Mendell has focused a lot of his research on neuronal plasticity, specifically within the [[Spinal cord|mammalian spinal cord]]. He has combined some of his research from NT-3 to help show how neurotrophins can help alter the function of both damaged and undamaged neurons.
* Pain
[[Pain]] is a physical feeling that most of the time harms an individual. This is caused by a damaging [[Stimulant|stimuli]] that can vary in intensity. The stimuli sends signals that then travel along neurons so that [[information]] can be received by the brain. The nerves that are typically responsible for pain sensation are [[Nociceptor|nociceptors]].
A lot of the work Mendell has done with both [[Neurotrophin|neurotrophins]] and [[Neuroplasticity|neuronal plasticity]] has some overlap into the realm of pain, and what their roles are in both reducing and relieving pain. For example, Mendell has conducted [[research]] which has shown the relationship between [[Nerve growth factor|nerve growth factors]] (a type of neurotrophin) and inflammatory pain.
* Nerve Wind Up
Mendell discovered the property of 'wind up' in the [[spinal cord]]. 'Wind Up' is the property of [[Group C nerve fiber|C-fibres]] in the [[Peripheral nervous system|peripheral nerves]] of the spinal cord that when stimulated at repetitious low frequencies there will be a gradual accumulation in the amplitude of response.<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref>
* NT-3
Mendell has been interested in the role [[Neurotrophin|neurotrophins]] play in nervous [[Neuroplasticity|plasticity]] since at least the mid 1990s. At the time, neurotrophins were understood to be significant factors in neuronal development and [[Differentiation (biology)|differentiation]] in the [[Central nervous system|central]] and [[peripheral nervous system]], however; scientists were still exploring the specific functions of individual neurotrophins. Subsequent findings suggested that neurotrophins have effects on both damaged and undamaged neurons. As such, neurotrophins are a major facet of [[spinal cord injury]] research. Dr. Mendell's research on neurotrophins has evolved synchronously with the understanding of neurotrophins. In more recent times, Dr. Mendell's research has been focused partly on spinal cord injury and the
* Nerve Growth Factor and Hyperalgesia
[[Hyperalgesia]], increase of sensitivity to a stimulus, was found by Mendell to be influenced by the introduction of [[nerve growth factor]], NGF. It functions by interactions with sensory [[Nociceptor|nociceptive]] neurons. Mendell was able to investigate how [[Rodent|rodents]] and [[Mammal|mammals]] sensitivity to thermal and mechanical stimulus was increased through exposure to NGF.<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref> NGF was shown by Mendell to also play a role in the development of young mammals. Mature animals, he showed, that NGF had a possible role in the linkage of [[hyperalgesia]] and [[inflammation]].<ref></ref>
== Major works/Contributions ==
In 1965, Mendell's doctoral dissertation was published in the academic journal [[Nature (journal)|Nature]]. The dissertation was about how [[Group C nerve fiber|C fibres]] in the dorsal horn affect the A fibres. He concluded that his evidence did not support the idea that the firing of C fibres had an effect on the presynaptic hyperpolarization of the A fibres of the cats that were experimented on.
In April 2016, Mendell helped to publish a paper<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref> that focused on the relationship between a protein called [[CD2AP]] and the plasticity of [[Neuron|neurons]]. It is known that the growth of an [[axon]] of a neuron helps with both the adaptive and maladaptive [[Neuroplasticity|plasticity]] within the [[nervous system]]. Therefore, if an axon becomes injured, or cannot function to its best ability, the consequences can be in the form of [[Neurological disorder|neurologic disease]]. The protein CD2AP in this study was looked at in depth, which is responsible for coordinating axon outgrowth. Mendell and his team were able to observe that CD2AP is amplified within [[Dorsal root ganglion|dorsal root ganglions]] (DRGs) during axonal sprouting, but decreases in quantity during axonal [[Regeneration (biology)|regeneration]]. Based on these findings, it is still undetermined what the exact relationship between the control of the regeneration of previously damaged axons and the additional development of non-damaged axons is.
In another one of his studies<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref>, conducted in July of 2011, Mendell et al. looked at the mechanisms involved in the relationship between [[Nerve growth factor|neuronal growth factors]] (NGF), which is a [[neurotrophin]], and pain. It is known that specific mutations in NGF or [[Tropomyosin receptor kinase A|TrkA]] genes can lead to a decrease in the sensitivity to [[pain]]. However, the NGF-TrkA axis, on the other hand, can facilitate the development of many different types of long-term acute and [[chronic pain]]. With this, this study was able to conclude that one has to look at how many NGF-responsive (meaning how many are TrkA-positive) [[Nociceptor|nociceptors]] are actually connected to the tissue where the pain is coming from. This is an important relationship because the amount of [[Nerve|innervation]] widely varies between different types of tissue. Then, once this has been examined, one can more effectively choose which chronic pain state to target. Therefore, if it is able to be applied effectively, choosing therapies that rely on targeting NGF-TrkA signaling may represent a new way to treat different types of chronic pain.
Mendell has also put forth research<ref>Liquid error: wrong number of arguments (given 1, expected 2)</ref> about the increased possibility of promoting [[spinal cord]] repair through a combination of several different [[Treatment|treatments]]. The mechanisms involved in this are due to the additive effects when several different neurotrophins and [[Protein|proteins]] are combined, including [[Chondroitin ABC lyase|chondroitinase ABC]], [[Neurotrophin-3|NT3]], and enhanced levels of [[NR2D]]. These together allow for either the synthesis or strengthening of spinal circuits, as well as slight recovery of function for those that have been damaged.
=== Selected Literature for continued reading on Mendell's Research. ===
**https://ift.tt/3bz3T88 (Abstract)
**https://ift.tt/33WsAch (Full Article)
**https://ift.tt/3dIVaCv (Abstract)
**https://ift.tt/2WWFRAc (Full Article)
**https://ift.tt/39u0ypB (Full Article)
**https://ift.tt/2QZRXV8 (Abstract)
**https://ift.tt/2UQV0jw (Abstract)
**https://ift.tt/2UHxFAM (Abstract)
**https://ift.tt/2UvQh8k (Full Article)
**https://ift.tt/2xz9UDe (Abstract)
**https://ift.tt/2WWO2ME (Abstract)
**https://ift.tt/2UHxGVm (Abstract)
**https://ift.tt/33W3yKe (Abstract)
**https://ift.tt/2Uyp58Z (Abstract)
**https://ift.tt/2JsLXQM (Abstract)
**https://ift.tt/2Jx1P4x (Abstract)
**https://ift.tt/2wP9M2u (Abstract)
**https://ift.tt/2WSvVrj (Full article)
**https://onlinelibrary.wiley.com/doi/abs/10.1111/j.1460-9568.2010.07392.x (Abstract)
**https://ift.tt/3dGNKPU (Full Article)
**https://ift.tt/3bH2OeS (Full Article)
**https://ift.tt/3bDamPt (Abstract)
**https://ift.tt/2vZo6F5 (Full Article)
**https://ift.tt/33ZHv5f (Full Article)
**https://ift.tt/2Uu2IRH (Full Article)
**https://ift.tt/2WSvWvn (Full Article)
**https://ift.tt/3augUQk (Abstract)
**https://ift.tt/39BuzUI (Full Article)
**https://ift.tt/39upCNa (Abstract)
**https://ift.tt/2QZ2joq (Abstract)
**https://ift.tt/2UNAd0p (Abstract)
**https://ift.tt/2ymTAWD (Full Article)
**https://ift.tt/2WTHv5u (Full Text)
**https://ift.tt/33XVXL7 (Full Article)
**https://ift.tt/3dISGnC (Abstract)
**https://ift.tt/39vWwx0 (Abstract)
=== Positions Held<ref> Renaissance School of Medicine at Stony Brook University|website=renaissance.stonybrookmedicine.edu|access-date=2020-03-29}}</ref> ===
Chair of the Department of Neurobiology and Behavior, [[Stony Brook University]], 1986-2006
[[Journal of Neurophysiology]]'s Editor-in-Chief, 1983-1989
On the Editorial Board for the [[Journal of Neurophysiology]], current
On the Editorial Board for the [[The Journal of Neuroscience|Journal of Neuroscience]], 1983-1989
On the Council for the [[Society for Neuroscience]], 1986-1990
President of the Association of [[Neuroscience]] Departments and Programs, 1991-1992
Treasurer of the [[Society for Neuroscience]], 1996-1997
President of the [[Society for Neuroscience]], 1997-1998
[[Board of directors|Board of Directors]] for the Craig Neilsen Foundation, 2009
Chairman for the [[Spinal Cord Injury|Spinal Cord Injury Research Board]] of the State of New York, 2013
Senior Editor for [[Neuroplasticity]] for the [[University of Oxford|Oxford University]] Press Research Encyclopedia in Neuroscience, 2015
=== Honors & Awards<ref> Renaissance School of Medicine at Stony Brook University|website=renaissance.stonybrookmedicine.edu|access-date=2020-03-29}}</ref> ===
Distinguished Professor of Neurobiology and Behavior at [[Stony Brook University]]
Selected to serve on the London (UK) Pain Consortium Advisory Board, 2009-2014
Chosen to give the [[History of neuroscience|History of Neuroscience]] plenary lecture ("The Emergence of Contemporary Pain Neuroscience") during the Annual Meeting of the Society for Neuroscience, 2012
Selected to be a part of the International Science Advisory Council of Brain Canada, 2014
Named a Fellow for the [[American Physiological Society]] Inaugural Class, 2015
<br />
March 31, 2020 at 12:43PM
